Drugs for the Control of Epilepsy: Actions on Neuronal Networks Involved in Seizure DisordersCRC Press, 4 июн. 2019 г. - Всего страниц: 558 Originally published in 1991. This book brings together the ideas of an international group of experts on clinical and experimental epilepsy. These authors consider how antiepileptic drugs may act on elements of neuronal networks to reduce seizure incidence and severity. The book addresses such topics as the four general classes of anticonvulsant drug mechanisms, major epilepsy models, the proposed mechanisms of action of major antiepileptic drugs, and the clinical use of antiepileptic drugs in the treatment of various forms of human epilepsy. This volume is special for its focus on the neuronal network approach to epilepsy, as well as for its comprehensive review and integration of human and animal data. Neurologists, pharmacologists, psychiatrists, and other investigators actively working on epilepsy research will find this book to be a useful, thought-provoking reference volume. |
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Drugs for the Control of Epilepsy: Actions on Neuronal Networks Involved in ... Carl L. Faingold,Gerhard H. Fromm Ограниченный просмотр - 2019 |
Drugs for the Control of Epilepsy: Actions on Neuronal Networks Involved in ... Carl L. Faingold,Gerhard H. Fromm Недоступно для просмотра - 2021 |
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abnormalities absence action activity administration agents agonists alterations amino acid animals antagonists anticonvulsant anticonvulsant drugs antiepileptic drugs appears associated audiogenic seizures barbiturates behavioral benzodiazepine binding block Brain Res brainstem calcium carbamazepine cells changes channels Chapter clinical cobalt complex concentrations convulsant cortex cortical decrease doses effects electroshock enhancement epilepsy epileptic epileptiform evidence excitability excitatory experimental Faingold Figure firing function GABA genetically GEPR gerbils glutamate half-life hippocampal important increase indicate induced inhibition inhibitory initiation injection interactions involved kindling levels major mechanisms Meldrum neuronal networks Neurosci neurotransmitter NMDA normal observed occur partial pathways patients Pharmacol phenobarbital phenytoin potential produced Raven Press receptor recorded reduced regions release repetitive reported response reticular role seizures severity shown similar slices sodium specific spontaneous stimulation structures studies suggested suppressed synaptic threshold treatment types valproate York