Molecular Biology of Alzheimer's Disease: Genes and Mechanisms Involved in Amyloid GenerationChristian Haass CRC Press, 2 сент. 2003 г. - Всего страниц: 330 Highlighting the latest and the most timely aspects of Alzheimer's disease research, this text will enable scientists in related research fields, as well as physicians working with Alzheimer's disease patients, to obtain a quick and complete overview of the current state of the art in one of the most exciting fields in neuroscience research. Leading scientists have contributed articles focusing on key developments in this field. This includes an overview about the pathology, the genetics of familial Alzheimer's disease, proteolytic generation and aggregation of amyloid -peptide, presenilins, risk factors such as ApoE, and transgenic animal models. Some of the latest developments in Alzheimer's disease research, including the effect of presenilin knock outs on amyloid -peptide generation, are also included. |
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Содержание
THE GENETICS OF FAMILIAL ALZHEIMERS DISEASE | 43 |
MOLECULAR AND CELLULAR BIOLOGY OF THE BAMYLOID PRECURSOR PROTEIN AND AMYLOID BPEPTIDE | 69 |
THE PRESENILIN GENES | 184 |
ROLE OF RISK FACTORS IN ALZHEIMERS DISEASE | 280 |
TRANSGENIC MODELS FOR ALZHEIMERS DISEASE | 297 |
321 | |
Другие издания - Просмотреть все
Molecular Biology of Alzheimer's Disease: Genes and Mechanisms Involved in ... Christian Haass Ограниченный просмотр - 2014 |
Molecular Biology of Alzheimer's Disease: Genes and Mechanisms Involved in ... Christian Haass Ограниченный просмотр - 2003 |
Molecular Biology of Alzheimer's Disease: Genes and Mechanisms Involved in ... Christian Haass Недоступно для просмотра - 1999 |
Часто встречающиеся слова и выражения
Acad activity addition allele alterations Alzheimer disease Alzheimer’s disease amino acid amyloid precursor protein analysis apoE apolipoprotein apoptosis appears associated binding biochemical Biol Biological brain cascade cause cell death cells changes Chem chromosome Citron cleavage compartment concentration containing cultured cytoplasmic deposits derived detected domain early effect elegans et al evidence expression factor familial Alzheimer’s disease fibril Figure formation fragments function gene genetic Haass human identified important increased indicate induced inhibition inhibitor interaction intracellular involved Journal kinase levels localization mechanisms membrane microtubule molecular mouse mutations Nature neurites neurons Neurosci normal observed occurs onset pathology pathway patients peptide phosphorylation plaques possible presenilin present Proc processing production protease receptor region regulation residues role Science secretion Selkoe sequence shown signal similar Sisodia specific Strooper structure studies suggest vivo